Eskenazi, Brenda; Chevrier, J; Rosas, LG; Anderson, HA; Bornman, MS; Bouwman, H; Chen, A; Cohn, BA; De Jager, C (May 4, 2009). "The Pine River Statement: Human Health Consequences of DDT Use". Environ. Health Perspect. 117 (9): 1359–1367. doi:10.1289/ehp.11748. PMC 2737010.
http://ehp.niehs.nih.gov/11748/
Might want to actually read it:
The recent literature shows a growing body of evidence that exposure to DDT and its breakdown product DDE may be associated with adverse health outcomes such as breast cancer, diabetes, decreased semen quality, spontaneous abortion, and impaired neurodevelopment in children.
Notice the word "may", as opposed to "will"
Conclusions: Although we provide evidence to suggest that DDT and DDE may pose a risk to human health, we also highlight the lack of knowledge about human exposure and health effects in communities where DDT is currently being sprayed for malaria control.
Again, "may".
Most human studies reviewed by IARC in 1991 did not show an association between DDT exposure and cancer risk.
Which seems to be what their study of studies reveals. but since they don't want to say that because everyone "knows" DDT is da ebil, we'll hedge our bets and hope no one wades into the weeds to actually read this.
They choose poorly.
Research on DDT/DDE exposure and cancer continued to yield mixed results after the publication of the IARC report.
Hmmm, "The evidence doesn't support our conclusions, but hey, we'll use weasel words instead."
Liver Cancer:
Blood levels of DDE were not associated with a higher risk.
DDE is the breakdown product of DDT.
How about Pancreatic Cancer:
In contrast, other studies have found no association between estimates of DDT exposure among workers (Cocco et al. 2005) or serum/ adipose tissue levels of p,p′-DDE and pancreatic cancer risk after adjustment for confounders (Hardell et al. 2007; Hoppin et al. 2000).
Breast Cancer !!! Yeah, everyone hates breast cancer:
Although neither DDT nor DDE induced mammary tumors in laboratory animal cancer bioassays (IARC 1991),
and
However, most case–control studies recently published and reviewed have not supported an association (Brody et al. 2004; Gatto et al. 2007; Ibarluzea et al. 2004; Iwasaki et al. 2008; Lopez-Cervantes et al. 2004; Siddiqui et al. 2005; Snedeker 2001).
What about maybe other cancers?
Research has not supported an association of DDT or DDE and incidence of colorectal, lung, bladder, prostate, endometrial, and stomach cancers (Baris et al. 1998; Cocco et al. 2005; Hardell et al. 2004; Howsam et al. 2004; Purdue et al. 2007; Sturgeon et al. 1998; Weiderpass et al. 2000). Although no associations were found with serum DDT (Rothman et al. 1997), higher DDE levels in dust, adipose tissue, and plasma have been associated with non-Hodgkin lymphoma in case–control studies (Colt et al. 2005; Quintana et al. 2004; Spinelli et al. 2007). For other cancers, such as leukemia (Flodin et al. 1988; Purdue et al. 2007), and testicular cancer (Hardell et al. 2006; McGlynn et al. 2008), evidence remains equivocal.
Maybe not.
Diebeetus:
Collectively, these studies from the United States and Sweden suggest that body burdens of DDT and/or DDE may be associated with the prevalence of diabetes. A variety of other persistent environmental chemicals also have been associated with diabetes prevalence (Lee et al. 2006). However, given the high correlation among various organochlorine exposures (Bradman et al. 2007), additional research is needed to delineate the specific contributions of DDT and DDE.
Vaskidmark and Wilford Brimley, call your lawyer.
Infants and chilluns !!!
However, two analyses of the CHDS, which was conducted around the same time as the CPP, have found no associations of DDT/DDE and preterm delivery or small for gestational age, despite slightly higher median DDE levels than in the CPP
Thus, associations between DDE levels and early weaning may be spurious, and further research is warranted.
The authors concluded that breast-feeding in itself may exert a protective effect against contaminants in human milk.
but found no associations between maternal serum p,p′-DDE and the odds of cryptorchidism, or between p,p′-DDT and p,p′-DDE concentrations and odds of hypospadias
Overall, the evidence for the relation of maternal DDT exposure and child physical growth is weak.
Fertility !!!
Overall, studies of highly exposed populations suggest that male fertility may be adversely affected by DDT exposure, but studies in populations with moderate to low exposure levels do not support a relationship between exposure and male fertility outcomes.
Overall, the few studies conducted to date suggest that DDT exposure may affect time to pregnancy, but more research is needed.
(I will note that some of the studies methodology is highly flawed. e.g. Using date of marriage to date of first pregnancy.)
I could go on, but
